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Yueli Yao

Yueli Yao

Arbeitsbereiche

Early Stage Researcher at the Helmholtz Zentrum München (GmbH), Institute of Epidemiology

Kontakt

Helmholtz Zentrum München
Ingolstädter Landstraße 1
D-85764 Neuherberg

Raum: 235a
Telefon: 01798297697

Project Title

Air pollution and health - what are the underlying biochemical mediators?

Research Interest

My research interests include the health effects of short-, median- and long-term exposure to air pollution on cardiovascular system, and mainly explore the underlying potential Biochemical mediators (omics biomarkers). Meanwhile, I'm also interested in the effect modifications and mediating factors (eg. nutrition and medication) in the association between air pollution and health outcomes.

Supervisors

Prof. Dr. Annette Peters
Prof. Dennis Nowak
Dr. Cavin K. Ward-Caviness
Dr. Alexandra Schneider

Curriculum Vitae

EDUCATION

 2019 - present Ph.D. candidate in Medical Research: Epidemiology & Public Health, LMU Munich, Germany
 2016 - 2019 Bachelor of Medicine, major in Environmental Toxicology, Lanzhou University, Lanzhou, China
 2011 - 2016 Bachelor of Medicine, major in Preventive Medicine, Lanzhou University, Lanzhou, China

Publications

1. Yueli Yao, Dong Wang, Haitao Ma, Chengyun Li, Xiaoru Chang, Patrick Lowc, S. Katharine Hammond, Mary Ellen Turyk, Junling Wang, Sa Liu. The impact on T-regulatory cell related immune response in rural women exposed to polycyclic aromatic hydrocarbons (PAHs) in household air pollution in Gansu, China: a pilot investigation. Environmental Research, doi: 10.1016/j.envres.2019.03.053 (2019).
2. Yueli Yao, Xiaoru Chang, Dong Wang, Haitao Ma, Huiling Wang, Haojun Zhang, Chengyun Li, Junling Wang. Roles of ERK1/2 and PI3K/AKT signaling pathways in mitochondria-mediated apoptosis in testes of hypothyroid rats. Toxicology Research, doi: 10.1039/c8tx00122g (2018).
3. Xiaoru Chang*, Yueli Yao*, Dong Wang, Haitao Ma, Panhong Gou, Chengyun Li, Junling Wang. Influence of hypothyroidism on testicular mitochondrial oxidative stress by activating the p38 mitogen-activated protein kinase and c-Jun NH2-terminal kinase signaling pathways in rats. Human Experimental Toxicology (joint first author) doi: 10.1177/0960327118781927 (2018).